Objective To study the mechanisms of protective effect of mild hypothermia on hepatic ischemia-reperfusion ( I-R) injury. 目的探讨亚低温对肝缺血再灌注损伤的保护作用机制。
Objective To study the function of circulating endothelium in lung with ischemia reperfusion. 目的探讨肺缺血再灌注(I-R)中内皮细胞功能的变化。
So the impaired mechanism of I-Rof limbs is of both theoretical and clinical importance. 因此,阐明肢体I-R引发海马神经元损伤的机制,具有重要的理论意义和临床应用价值。
Objective To quantitatively analyse the imaging of myocardial ischemia-reperfusion ( I-R) injury using leukocyte-targeted contrast echocardiography with ultrasound tissue characterization. 目的用超声组织定征量化分析白细胞靶向超声造影剂对犬心肌缺血再灌注(ischemia-reperfusion,I-R)损伤声像图。
As those two factors correlate closely with energy metabolism and former researches have disclosed the adverse change of their levels in metabolic conditions, we hypothesized that their levels might undergo a certain degree of fluctuation in the severe metabolic disorders of intestinal I-R injury. 由于它们都与能量代谢密切相关,而且以往的研究发现其浓度变化成相反的趋势,我们假设它们在肠I-R损伤所致的能量代谢障碍中发生一定的变化。
Objective: To explore the effects of cytokines on renal ischemia reperfusion ( I R) injury. 目的:通过观察大鼠肾脏缺血再灌注(I-R)的不同时间IL-6和TNF-α的变化,探索细胞因子在肾脏I-R损伤中的作用。
Objective To investigate the effect of pulmonary injury after intestinal ischemia reperfusion ( I-R), and the mechanism of methylene blue ( MB) to radical oxygen species ( ROS). 目的观察肠缺血再灌注(I/R)后对肺脏的影响,探讨亚甲蓝(MB)的抗自由基(ROS)损伤作用及机制。
The possibility of using red blood cells ( RBC) as the carrier of superoxide dismutase ( SOD) was studied in this paper. SOD was encapsulated into rabbit erythrocytes ( RBC-SOD), and was iised in the study of cerebral ischemiareperfusion ( I-R). 研究用红细胞(RBC)作为超氧化物歧化酶(SOD)载体的可行性,将包载SOD的RBC(RBC-SOD)用于家兔脑缺血-再灌注(I-R)模型研究。
Sixty adult male Wistar rats were randomly allocated to sham operation group ( Sham), ischemia-reperfusion group ( I-R) and propofol group with 20 animals each. 60只雄性Wistar大鼠,随机分为假手术组(Sham组)、缺血-再灌注组(I-R组)和丙泊酚组(P组),每组20只。
The S-T segment elevation ( behind point J 0.04 second) induced by I-R in rabbits. 缺血/再灌注期间心电图上ST段上抬幅度(J点后0.04秒测量)。
On rat in situ perfused intestine, it was found that PAF released from intestinal tissue was significantly increased during ischernia-reperfusion injury of the intestine. 在大鼠小肠原位灌注模型上,发现小肠缺血&再灌注(I-R)损伤时,肠道产生的PAF显著增加;
Conclusion: I-R can increase the expression of cell adhesion molecules and cell chemotactic factor. 结论:I-R促进大鼠细胞黏附分子、趋化因子的表达;
RESULTS: HE staining showed penumbra neuron damages were alleviated significantly in prevention group, I R treatment group and post reperfusion treatment group as compared with those of I R group. 结果:苏木精-伊红染色示预防组,I-R治疗组,再灌注后治疗组较之I-R组缺血半暗区神经元损伤明显减轻。
Conclusion it is proved in IPK model that the generation of ONOO~ (-) possesses important role in renal vascular response damage of rats after renal I-R. 结论ONOO-在肾I-R所致的肾血管反应性损伤中具有重要的作用。
Myocardial I-R injury was quantitive analysed by ultrasound tissue characterization. 超声组织定征定量分析缺血再灌注损伤心肌。
Intestinal ischemia-reperfusion ( I-R) injury is a classical model reflecting acute stress and inflammatory responses, during which process the body suffers severe metabolic disorders and out-of-control inflammatory responses. 肠缺血-再灌注(ischemia-reperfusion,I-R)损伤是一种反映急性应激及炎症反应的模型,在损伤过程中机体出现严重的能量代谢障碍和失控炎症反应。
The anti-proliferative effect of agmatine on tumor cells didn't due to the activation of I-R and its cell toxicity. 肌丁胺抑制肿瘤增殖的作用与激活咪哇琳受体和细胞毒作用无关。
Conclusion After the intestinal I-R, the different clinical doses of propofol have protective effects on the lungs against intestinal I-R-induced injury. 结论小肠I-R后,不同临床剂量异丙酚可以不同程度地减轻肺组织损伤。
3.1. Compared with sham operation group, the activity of SOD was significantly lower but MDA contents were significantly higher in I-R group ( PO. 01). 3.1与假手术组相比,缺血一再灌注组脑组织中SOD活性显著降低(P<0.01),MDA含量显著升高(P<0.01)。
These results indicate that intact RBC-SOD could scavenge superoxide anion() producedduring cerebral I-R. 提示完整的RBC-SOD载体能够清除脑I-R时产生的超氧阴离子自由基()。
Anisodamine added in the perfusion fluid obviously inhibited the PAF release, and also showed anti-I-R injury effect, these effects were similar to that of PAF receptor antagonist Kadsurenone. 山莨菪碱灌流明显抑制其PAF的产生,并有与PAF受体拮抗剂Kadsurenone相似的抗I-R损伤效应。
The injury degree of pancreas graft in group SF was more severe than in group I-R. I-R组移植胰的损伤程度大于SF组。
Objective To research the functions of different clinical doses of propofol on the lung injury induced by intestinal ischemia-reperfusion ( I-R) in rats. 目的研究不同临床剂量异丙酚对大鼠小肠缺血再灌注(I-R)中肺损伤的作用。
Inhibition of HO-1 activity induced more severe brain injury in I-R group. 抑制HO-1活性,I-R组脑组织损伤加重。
In vivo models of myocardial ischemia-reperiusion ( I-R) and ischemic preconditioning ( IPC) were established with adult SD rats. 用成年SD大鼠建立在体心肌缺血&再灌注(I-R)模型及缺血预处理(IPC)模型。
Healthy male Wistar rats were randomly divided into sham group, ischemia – reperfusion ( I-R) group, NBP treated group, each 18. 健康雄性Wistar大鼠随机分成假手术组、缺血再灌注组、NBP(丁苯酞)干预组,每组18只。