hypersecretion

1. Airway Mucus Hypersecretion in Rats Is Inhibited by the Extracts of Pinellia
   半夏提取物对气道黏液高分泌的影响
2. Mucus hypersecretion in airway is the common pathophyisolo gi cal characteristic of many chronic airway inflammation diseases.
   目的：介绍慢性炎症气道粘液高分泌的治疗进展。
3. Roles of theaflavins in inflammatory airway mucus hypersecretion
   红茶提取物在炎性气道黏液高分泌中的作用
4. Cigarette smoking increases the irritability of the small airways, causes mucus hypersecretion, and decreases mucociliary transport.
   吸烟增加小气道的反应性，使粘液分泌过多，降低粘膜-纤毛转运。
5. Objective To explore the effect of simvastatin on airway mucus hypersecretion in rats.
   目的了解辛伐他汀对丙烯醛引起的气道黏液高分泌的作用及其可能的机制。
6. Objective To investigate the effects of macrolide on and the role of nuclear factor-κ B ( NF-κ B) in airway mucus hypersecretion stimulated by acrolein in rats.
   目的探讨大环内酯类抗生素对丙烯醛诱导的大鼠黏液高分泌模型的干预作用，以及核因子-κB（NF-κB）在黏液高分泌发病机制中的作用。
7. Objective To compare the inducing effects of different secretagogues on hypersecretion of respiratory mucosa.
   目的比较不同因素对气道黏液高分泌的诱导作用，为动物模型制造和临床防治提供指导。
8. Somatostatin analogs, such as octreotide, successfully control hormone hypersecretion in patients with acromegaly, islet cell tumors, carcinoids, and control of tumor growth.
   生长抑素类似物奥曲肽在肢端肥大症和良、恶性腺细胞肿瘤的患者中能成功地控制激素的高分泌，控制肿瘤的生长。
9. Effect of p38 MAPK and MMP-9 signaling pathway on airway mucus hypersecretion
   p38MAPK与MMP-9在气道粘液高分泌中的调控作用
10. Mechanism of Chronic Airway Inflammation Mucous Hypersecretion Inhibited by Macrolides
    大环内酯类抗生素对慢性气道炎症黏液高分泌的干预机制
11. Effect of Eucalyptus globulus oil on lipopolysaccharide-induced chronic bronchitis and mucin hypersecretion in rats
    蓝桉油对脂多糖引起的大鼠慢性支气管炎及黏蛋白高分泌的影响
12. The expression and significance of nuclear factor-κ B in the airway of rats with mucous hypersecretion
    核转录因子-κB在慢性气道粘液高分泌大鼠肺组织中的表达及意义
13. A comparative study on airway local therapy inhibiting airway mucous hypersecretion
    气道局部治疗对气道黏液高分泌干预作用的对比研究
14. Conclusion Rosiglitazone can decrease the expressions of TNF-α, IL-8 and suppress mucus hypersecretion in rat airway.
    结论罗格列酮能够下调TNF-α和IL-8的表达，减轻气道黏液高分泌。
15. Background and Objective Asthma is chronic airway immune inflammation characterized by airway lymphocyte infiltration and eosinophilia, airway hyperresponsiveness ( AHR) and mucus hypersecretion.
    背景与目的哮喘是慢性气道免疫性炎症，以气道淋巴细胞、嗜酸性粒细胞浸润、气道高反应性、粘液产生增加为特征。
16. Research progress of airway mucus hypersecretion in asthma
    哮喘黏液高分泌研究进展
17. Conclusion ( 1) OA pathogenesis is related to hypersecretion of cytokines and their release into blood.
    结论（1）OA的发病机制与关节滑膜细胞分泌过多的细胞因子以及这些细胞因子释放入血有关。
18. The changes of the brain function and cerebral blood flow dynamics are closely correlated with the thyroxin hypersecretion.
    脑功能和脑血流动力学的改变与甲状腺激素分泌过多有关。
19. Conclusion: The increased leukocyte adhesiveness and hypersecretion of TNF is probably one of the mechanisms which take part in the pathogenesis of lung injury in ANP.
    结论：白细胞粘附增强和TNF的过度分泌可能是ANP肺损伤发生的机制之一。
20. Chronic inflammatory processes of the respiratory system such as COPD, asthma and pulmonary cystic fibrosis are characterised by airway disorganization, reconstruction and mucous hypersecretion.
    在慢性阻塞性肺疾病、哮喘和肺囊性纤维化等气道慢性炎症性疾病中，常伴有广泛的气道结构破坏、重构和黏液高分泌等病理改变。
21. Influence of rosiglitazone on mucus hypersecretion in rat airway epithelium
    罗格列酮对大鼠气道黏液高分泌的影响
22. It is suggested that DU is probably caused by the hypersecretion of gastric acid, and GU by the deteriorated function of the gastric mucosal barrier.
    提示：DU和GU发生机制不同，DU的发生可能与高胃酸分沁量有关，而GU则可能由胃粘膜屏障机能减退所致。
23. Conclusion: Ber can reversed intestinal hypersecretion induced by ChT.
    结论：Ber可抑制由ChT所效的电解质分泌亢进。
24. To investigate the molecular mechanisms of mucous hypersecretion inhibited by macrolides in chronic inflammatory airway.
    为了解大环内酯抗生素抑制慢性炎症气道粘液高分泌的分子机制。
25. The results suggest that the hypersecretion of cortisol might be closely related to the non-stress peptic ulcer as well.
    然昼夜分泌节律相仿。结果提示，非应激性消化性溃疡有皮质醇分泌的明显异常。
26. For pituitary hormone of PRL and GH, hypersecretion state improved a lot after the operation.
    对于PRL及GH型的垂体腺瘤激素高分泌状态术后得到改善。
27. Objectives: Studies have shown that hypersecretion and increased glycosylation levels of mucus and mucins occur in the processes of airway acute and chronic inflammation.
    目的：气道急性和慢性炎症过程中粘液及粘蛋白高分泌，糖基化程度也显著增加。
28. In our research, we set up the models of SD rats chronic airway inflammation, and observe the effect of eucalyptus globulus oil on chronic airway inflammation and mucin hypersecretion.
    故在本研究中，我们建立了SD大鼠慢性气道炎症模型，并观察了蓝按油在气道炎症和粘蛋白高分泌中的作用。
29. Gastric hypersecretion might damage the mucus-bicarbonate barrier and the mucosal barrier.
    胃酸过多可以损伤黏液-碳酸氢盐屏障和粘膜屏障。

noun

1. excessive secretion