The hydrogen peroxide eliminates by the catalase and the peroxide enzyme, the oxygen free eliminates fragrant by the hyperoxide mutase. 过氧化氢由过氧化氢酶和过氧化物酶清除,氧自由荃由超氧化物歧化酶清除。
Results Hypothermic brain protection could considerably reduce 60% lipid hyperoxide in brain tissues as compared with that in the control group. It could also decrease heart rate and respiration and hence reduce tissue oxygen consumption. 结果与对照组相比,低温脑保护仪能降低脑组织60%脂质过氧化物的产生,减缓心率和呼吸,从而降低氧耗。
The contents of elements Cu and Fe all declined significantly. the two elements had something to do with the activity reducing of peroxidase ( POD) and hyperoxide dismutase ( SOD) in the host body. Cu、Fe的含量都有较明显的降低、这两种元素与寄主体内过氧化物酶(POD)、超氧化物歧化酶(SOD)的活性降低有关。
Conclusion: The technique was simple and reliable, better repetitive, and helped to understand anti hyperoxide lipid of human body in cellular level. 结论:该方法简单可靠,重复性好,并可从细胞水平上了解机体的抗脂质过氧化作用。
Conclusions The results suggest that CGRP has effects of protecting the myocardial injury induced by isoproterenol, correcting the imbalance between MDA and SOD level, inhibiting the stronger lipid hyperoxide reaction, preventing the myocardial enzyme leak from injuried cardiomyocyte and antagonizing arrhythmia in rats. 结论静脉用CGRP治疗异丙肾上腺素所致大鼠心肌损伤,能纠正MDA和SOD的失衡,抑制过强脂质过氧化反应,并保护心肌细胞,防止心肌酶漏出,拮抗心律失常。
Akt can accommodate the activity of nitric oxide synthase, and increase the synthesis of nitric oxide, and inhibit platelet aggregation and its combining with hyperoxide, and prevent hepatocytes from damage caused by oxygen free radical. Akt能够调节NOS的活性、进而增加N0的合成,抑制低血流量下的血小板聚集并与超氧化物结合,形成无毒代谢产物NO3-;从而使肝细胞避免氧自由基所致损害,保护肝细胞。